Hematology

G6PD (Glucose-6-Phosphate Dehydrogenase) — What Your Blood Test Result Means

ScanHealth Learn Hematology G6PD (Glucose-6-Phosphate Dehydrogenase)

Your Red Blood Cell Defense Enzyme

G6PD is an enzyme that protects red blood cells from oxidative damage. It generates NADPH, which is the antioxidant shield for RBCs. Without enough G6PD, certain medications, foods (fava beans), or infections cause oxidative stress that destroys red blood cells—hemolytic anemia. It's the most common enzyme deficiency worldwide, affecting ~400 million people.

What is G6PD (Glucose-6-Phosphate Dehydrogenase)?

G6PD is the rate-limiting enzyme in the pentose phosphate pathway, generating NADPH for glutathione reduction (antioxidant defense in RBCs). X-linked recessive: primarily affects males. ~400 million people worldwide are deficient. Testing during acute hemolysis may be falsely normal (deficient cells already destroyed).

What High G6PD (Glucose-6-Phosphate Dehydrogenase) Means

Not clinically significant.

Common symptoms:

Not applicable

What Low G6PD (Glucose-6-Phosphate Dehydrogenase) Means

G6PD deficiency—your red blood cells are vulnerable to oxidative stress. Triggers (medications, fava beans, infection) can cause sudden, severe hemolytic anemia. X-linked inheritance: primarily affects males. Very common in Mediterranean, African, and Asian populations.

Common symptoms:

During hemolytic crisis: sudden anemia, fatigue, pallor, jaundice, dark urine · Back pain (from massive hemolysis) · Shortness of breath, tachycardia · Between crises: completely asymptomatic

Why It Matters

When normal:

Identifies susceptibility to drug-induced hemolysis

Prevents life-threatening hemolytic crises

Guides medication safety (avoid oxidant drugs)

One-time test (genetic condition)

Risks if abnormal:

Deficient: hemolytic crisis with oxidant exposure

Medications to avoid: primaquine, dapsone, rasburicase, methylene blue, sulfonamides, nitrofurantoin

Fava beans can trigger severe hemolysis ("favism")

What Can Cause Abnormal Levels?

Genetic (X-linked)

100% likely

Inherited enzyme deficiency. Over 400 genetic variants identified. Severity varies by variant.

Testing Timing

Testing during acute hemolysis gives falsely normal results (deficient cells already hemolyzed). Retest 2-3 months after acute episode.

What You Can Do

AVOID oxidant medications: primaquine, dapsone, rasburicase, nitrofurantoin, sulfonamides

Impact: Prevents hemolytic crisis \u00B7 Timeline: Lifelong

Avoid fava beans (broad beans) if severe deficiency

Impact: Fava beans contain oxidants that trigger hemolysis \u00B7 Timeline: Lifelong

Carry medical alert identification

Impact: Ensures healthcare providers know to avoid oxidant drugs \u00B7 Timeline: Always

If lifestyle changes aren't enough:

If hemolytic crisis occurs: supportive care, hydration, transfusion if severe

Impact: Most episodes are self-limited once trigger is removed \u00B7 Timeline: Acute

Recommended retest: One-time genetic test (but retest if drawn during acute hemolysis)

Related Markers

hemoglobin reticulocyte_count haptoglobin ldh bilirubin_indirect
Medical Disclaimer: This information is for educational purposes only and is not a substitute for professional medical advice. Always consult your doctor for diagnosis and treatment.

Got your blood test report?

Upload your PDF and understand ALL your markers in 2 minutes. Plain language. Traffic light status. No medical jargon.

Analyze My Report — Free

First report is free. No credit card needed.

Browse all markers